As with any trial and error testing, there must of course be a starting point and an endpoint where data can be accumulated and tested. While there is nothing in the literature citing specifically why 90 days has been established as a testing period, as in any testing protocol a baseline must be established and data collected over a period of time. This generally results in changes over a period of time during the testing period. But at some point there the law of diminishing returns sets in and additional testing results becomes insignificant to a study’s outcome. It is logical to assume that 90 days has become that standard period of testing in animal feeding studies. According to EFSA (European Food Safety Authority), “90-day animal feeding studies are often used to provide information for the risk assessment of food and feed and/or of individual substances contained therein.”
A recently published research paper in the journal New Biotechnology suggested that longer term testing rather than the classical 90-day studies be performed on a case by case basis, and should not be the norm. The authors concluded that “long-term and multigenerational studies should only be conducted in a case-by-case approach for GE food/feed safety and nutritional regulatory assessment if some reasonable doubt remains after the 90-day rodent feeding trial.” In fact, based on their research none of these longer-term assessments “have raised new safety concerns [regarding] marketed GE crop varieties.” They conclude that the data “does not provide evidence that more food safety testing is necessary for GE crop varieties.” In fact they claim that longer term multigenerational data could actually lower the risk assessment of GE crop varieties. And finally, the researchers proclaimed that governments are trying to “demonstrate environmental risks for cultivation of GE crops” which “fail to provide scientifically valid data.”
Additionally, I am not in agreement that it is universally accepted that “disease and pathology often takes many months, sometimes years to develop.” Changes in cell structure and blood cells occur rather quickly when exposed to pathogens. This effect should not be confused with the diagnosis of such cellular abnormalities which are oftentimes not discovered for possibly months or years later when symptoms from these mutations actually begin to surface.